Herbs (Global Traditional) · Native American
Black Haw (Viburnum prunifolium) (Viburnum prunifolium)
Moderate Evidencebotanical
Hermetica Superfood Encyclopedia
Black haw (Viburnum prunifolium) is a North American shrub containing salicin and scopoletin that acts as a uterine antispasmodic. These compounds work by relaxing smooth muscle tissue, particularly in the uterus, making it traditionally valued for menstrual support.
Black haw is a shrub native to eastern North America, particularly the woodlands of the southern United States, with medicinal components derived from the root bark and inner bark of Viburnum prunifolium. The herb is traditionally prepared as tinctures, decoctions, teas, and topical creams or lotions from the dried bark.
Clinical trial data for black haw is severely limited, with WebMD stating there is no good scientific evidence to support traditional uses. The only available evidence comes from in vitro studies showing scopoletin inhibits COX-2 synthesis and animal studies or anecdotal reports. No human randomized controlled trials, meta-analyses, or PubMed-indexed clinical studies are referenced in the available sources.
Traditional preparation: Bark tea - 1 tablespoon of dried bark steeped in 8 ounces of water for 10 minutes. Tinctures are applied for muscle relaxation or menstrual pain but specific dosing is not standardized. No clinically studied dosage ranges or standardized extract concentrations are available. Consult a healthcare provider before starting any new supplement.
Black Haw (Viburnum prunifolium) bark and root bark are not consumed as a food source, so macronutrient profiling is not applicable in conventional dietary terms. However, the plant contains a well-characterized set of bioactive compounds: Scopoletin (6-methoxy-7-hydroxycoumarin) is the primary documented constituent, present at approximately 0.3–0.5% dry weight in the bark, functioning as a smooth muscle relaxant and mild COX-2 inhibitor. Salicin and related salicylates (including salicosides) are present at roughly 0.1–0.3% dry weight, contributing mild analgesic and anti-inflammatory activity similar in mechanism to aspirin precursors. Isovaleric acid (3-methylbutanoic acid) has been identified in the volatile fraction, historically thought to contribute to antispasmodic effects, though its concentration is low (<0.1%). Triterpenoids including oleanolic acid and ursolic acid are present in small quantities in the bark (~0.05–0.15%). Tannins (primarily condensed tannins/proanthocyanidins) are present at approximately 2–4% dry weight, contributing astringent properties. Flavonoids including amentoflavone and chlorogenic acid have been detected in small amounts (<0.1%). No significant vitamins, dietary minerals, dietary fiber, or protein content is documented, as the bark is used exclusively in extract or tincture form, typically at doses of 1–2 mL tincture (1:5 ethanol extract) or 2–4 g dried bark in decoction. Bioavailability of scopoletin from oral preparations is estimated to be moderate, with hepatic first-pass metabolism producing active glucuronide conjugates. Salicylate bioavailability from salicin conversion occurs via gut bacterial hydrolysis followed by hepatic oxidation.
Black haw's primary bioactive compounds salicin and scopoletin inhibit prostaglandin synthesis and modulate calcium channels in smooth muscle cells. Salicin converts to salicylic acid, reducing inflammatory prostaglandins that cause uterine contractions. Scopoletin acts as a smooth muscle relaxant by blocking calcium influx and reducing muscle fiber excitability.
No randomized controlled trials have been conducted on black haw specifically. Traditional use studies and case reports suggest effectiveness for menstrual cramps, but these lack control groups and standardized dosing. Most evidence comes from historical Native American use documentation and European folk medicine records. The absence of clinical trials means efficacy claims rely entirely on traditional evidence and theoretical mechanisms.
Black haw may interact with anticoagulant medications due to its salicin content, which has blood-thinning properties. Pregnant women should avoid use as it may stimulate uterine contractions despite its antispasmodic reputation. Common side effects include gastrointestinal upset and potential allergic reactions in salicylate-sensitive individuals. Those taking aspirin or other NSAIDs should consult healthcare providers before use.
